Tag: Neuroprotection

The human body maintains a constant core temperature, but when it drops below 35°C (95°F), a state known as hypothermia begins. Understanding the precise mechanism of action of hypothermia reveals how this potentially life-threatening condition alters metabolic processes and organ function at a cellular level, sometimes with life-saving results.

According to the University of Rochester Medical Center, therapeutic hypothermia is a clinical treatment used to moderately lower a patient's body temperature. A key piece of equipment for this procedure is a cooling blanket, used in hospital settings for various critical medical applications to control and reduce a patient's body heat.

Temperature management, specifically Targeted Temperature Management (TTM), is a controlled medical intervention to regulate a patient's body temperature for therapeutic purposes. Often used in critical care settings following events like cardiac arrest, this therapy helps mitigate cellular damage and improve patient outcomes.

In critical care, precise thermal control is crucial for patient outcomes; for example, targeted temperature management (TTM) is used post-cardiac arrest to improve neurological function. This practice, known as **temperature management for patients**, involves regulating body temperature to aid healing and protect vital organs.

Medical studies have consistently shown that controlling body temperature after a traumatic event can significantly improve patient outcomes. The procedure in which body temperature is lowered for medical reasons is a critical intervention used to reduce neurological damage and protect vital organs.

According to the American Heart Association, targeted temperature management (a type of induced hypothermia) is a recommended treatment for comatose survivors of out-of-hospital cardiac arrest. The procedure is a carefully controlled process that involves lowering a patient's core body temperature to specific ranges to reduce cell damage and improve outcomes.

Did you know that cotinine has a significantly longer half-life in the body than nicotine itself? This makes it the most reliable biomarker for tracking tobacco exposure. Understanding what is the function of cotinine is crucial for gauging the lingering effects of nicotine intake and secondhand smoke.

In preclinical animal studies, a specific Smoothen agonist, commonly referred to as SAG therapy, has shown remarkable results, such as restoring bone growth and decreasing mortality in models of skeletal dysplasia. This highlights its potential as an innovative, pathway-based treatment under investigation.